1. Robert L. Myette, Fengxia Xiao, Pavel Geier1, Janusz Feber1, Dylan Burger and Christopher R. J. Kennedy. Urinary podocyte-derived large extracellular vesicles are increased in paediatric idiopathic nephrotic syndrome. Nephrol Dial & Transplantation. 2023; 38, 2089-91.
2. Jha, J.C., Dai, A., Garzarella, J., Charlton, A., Urner, S., Østergaard, J.A., Okabe, J., Holterman, C.E., Skene, A., Power, D.A., Ekinci, E.I.,Coughlan, M.T., Schmidt, H. H.W., Cooper, M.E., Touyz, R.M., Kennedy, C.R.J., Jandeleit-Dahm, K. Independent of Renox, NOX5 Promotes Renal Inflammation and Fibrosis in Diabetes by Activating ROS-Sensitive Pathways. Diabetes 2022; 71, 1282–1298.
3. Holterman CE, Boisvert NC, Thibodeau JF, Kamto E, Novakovic M, Abdelrahman K, Ferguson S, Kennedy CRJ. Podocyte NADPH Oxidase 5 promotes renal inflammation regulated by the toll-like receptor pathway. Antioxid Redox Signal. 2019;30(15):1817-1830.
4. Holterman CE, Boisvert NC, Thibodeau J-F, Kamto E, Novakovic M, Abdelrahman K, Ferguson S, Kennedy CRJ. Podocyte NADPH Oxidase 5 promotes renal inflammation regulated by the toll-like receptor pathway. Antioxid Redox Signal. 2018;doi.org/10.1089/ars.2017.7402.
5. Boisvert NC, Holterman CE, Gutsol A, Coulombe J, Pan W, Alexander RT, Gray DA, Kennedy CRJ. Ubiquitin COOH-terminal hydrolase L1 deletion is associated with urinary α-klotho deficiency and perturbed phosphate homeostasis. Am J Physiol Physiol. 2018;315(2):f353-363.
6. Boisvert NC, Holterman CE, Thibodeau J-F, Nasrallah R, Kamto E, Comin CH, da F. Costa L, Carter A, Hébert RL, Gutsol A, Cron GO, Lacoste B, Gray DA, Kennedy CRJ. Hyperfiltration in Ubiquitin C-terminal hydrolase L1 -deleted mice. Clin Sci. 2018;132:1453-1470.
7. Boisvert NC, Holterman CE, Gutsol A, Coulombe J, Pan W, Alexander RT, Gray DA, Kennedy CRJ. Ubiquitin C-terminal hydrolase L1 deletion is associated with urinary α-klotho deficiency and perturbed phosphate homeostasis. Am J Physiol Physiol. 2018;315(2):F353-F363. http://www.ncbi.nlm.nih.gov/pubmed/29667913
8. Thibodeau JF, Holterman CE, He Y, Carter A, Cron GO, Boisvert NC, Abd-Elrahman KS, Hsu KJ, Ferguson SS, Kennedy CRJ. Vascular smooth muscle-specific EP4 receptor deletion in mice exacerbates angiotensin II-induced renal injury. Antioxid Redox Signal. 2016;25(12):642-656.
9. Burger D, Thibodeau JF, Holterman CE, Burns KD, Touyz RM, Kennedy CRJ. Urinary podocyte microparticles identify prealbuminuric diabetic glomerular injury. J Am Soc Nephrol. 2014;25(7): 1401-1407.
10. Holterman CE, Thibodeau JF, Towaij C, Gutsol A, Montezano AC, Parks RJ, Cooper ME, Touyz RM, Kennedy CRJ. Nephropathy and elevated BP in mice with podocyte-specific NADPH oxidase 5 expression. J Am Soc Nephrol. 2014;25(4):784-797.
11. Read NC, Gutsol A, Holterman CE, Carter A, Coulombe J, Gray DA, Kennedy CRJ. Ubiquitin C-terminal hydrolase L1 deletion ameliorates glomerular injury in mice with ACTN4-associated focal segmental glomerulosclerosis. Biochim Biophys Acta. 2014;1842(7):1028-1040.
12. Thibodeau JF, Holterman CE, Burger D, Read NC, Reudelhuber TL, Kennedy CRJ. A novel mouse model of advanced diabetic kidney disease. PLoS One. 2014;9(12):e113459.
13. Thibodeau JF, Nasrallah R, Carter A, He Y, Touyz RM, Hébert RL, Kennedy CRJ. PTGER1 Deletion Attenuates Renal Injury in Diabetic Mouse Models. Am J Pathol. 2013;183(6):1789-802.
14. Stitt-Cavanagh EM, Faour WH, Takami K, Carter A, Vanderhyden B, Guan Y, Schneider A, Breyer MD, Kennedy CRJ. A maladaptive role for EP4 receptors in podocytes. J Am Soc Nephrol. 2010; 21(10):1678-90.